The Journal of the American Dental Association
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J Am Dent Assoc, Vol 137, No 11, 1497.
© 2006 American Dental Association

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LETTERS

Authors’ response

We agree that the bisphosphonate-induced osteonecrosis of the jaw (ONJ) exhibited in this patient was most likely a result of the periodontal procedure, due to the location of the sequestrum and clinical observations. However, previous reports in the endodontic literature1,2 present cases of bisphosphonate-induced ONJ in which clinical findings of normal, or only slightly altered, periradicular tissue before endodontic treatment resulted in ONJ. We believe, albeit rare, that these parallels justify the inclusion of a potential etiology of the endodontic procedure in our report.


   REFERENCES
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 REFERENCES
 
  1. Sarathy AP, Bourgeois SL Jr, Goodell GG. Bisphosphonate-associated osteonecrosis of the jaws and endodontic treatment: two case reports (published correction appears in J Endod 2005;31[11]:835–6). J Endod 2005;31(10):759–63.[Medline]

  2. Katz, H. Endodontic implications of bisphosphonate-associated osteonecrosis of the jaws: a report of three cases. J Endod 2005;31(11):831–4.[Medline]



John B. Nase, DDS, Associate Professor of Restorative Dentistry (Adjunct)

School of Dentistry

Jon B. Suzuki, DDS, PhD, MBA, Professor and Associate Dean for Graduate Education

Research and International Relations, Temple University Philadelphia



This Article
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